Sex differences in viral load and disease progress

This article is more than 24 years old.

Since last November’s report that the relationship between

viral load and disease progression in women may not be the same as in men,

clinicians and activists have been questioning whether women should be

Glossary

disease progression

The worsening of a disease.

equivalence trial

A clinical trial which aims to demonstrate that a new treatment is no better or worse than an existing treatment. While the two drugs may have similar results in terms of virological response, the new drug may have fewer side-effects, be cheaper or have other advantages. 

replication

The process of viral multiplication or reproduction. Viruses cannot replicate without the machinery and metabolism of cells (human cells, in the case of HIV), which is why viruses infect cells.

hormone

A chemical messenger which stimulates or suppresses cell and tissue activity. Hormones control most bodily functions, from simple basic needs like hunger to complex systems like reproduction, and even the emotions and mood.

boosting agent

Booster drugs are used to ‘boost’ the effects of protease inhibitors and some other antiretrovirals. Adding a small dose of a booster drug to an antiretroviral makes the liver break down the primary drug more slowly, which means that it stays in the body for longer times or at higher levels. Without the boosting agent, the prescribed dose of the primary drug would be ineffective.

encouraged to start treatment at lower viral loads than men.

Rates of disease progression at different viral load and CD4

levels were first calculated after an analysis of the MACS cohort of gay men, a

group followed since 1982. These findings formed the basis for US and

international guidelines on when to start treatment.

Homayoon Farzedegan and colleagues at Johns Hopkins School

of Public Health (The Lancet Nov 7 1998

href="http://www.thelancet.com">http://www.thelancet.com

) found that women

had a 60% higher risk of developing AIDS than men at the same viral load level.

The study looked at viral load differences between 527 male and female injecting

drug users tested in 1988 and 285 tested in 1993. The average follow-up was

almost six years. In other words, women with viral load of 20,000 copies were

1.6 fold more likely to develop AIDS in the follow-up period than men with

similar viral load.

Subsequently, colleagues at Johns Hopkins University School

of Medicine (The Lancet February 6 1999

href="http://www.thelancet.com">http://www.thelancet.com

) reported that they

found no difference between men and women in viral load and rates of disease

progression.

Now, several other research groups have looked into disease

progression rates amongst their own patients. Swiss and Italian researchers have

failed to find any difference between men and women. The Swiss HIV Cohort Study

looked at 1337 men and women, and found no difference in viral load levels at a

range of CD4 counts, nor any difference in the risk of developing AIDS during

the follow up period. In the Italian ICONA cohort, men and women had similar

viral load levels when they were matched according to their CD4 counts, unlike

in the Farzedegan study. The ICONA study reported on 2011 men and women (The

Lancet, February 13 1999

href="http://www.thelancet.com">http://www.thelancet.com

).

Explanations for the discrepancy spotted by Johns Hopkins

School of Public Health are far from clear. Hormonal difference have been

suggested, but a study by University College School of Medicine in 1995 found

that HIV-negative women patients in a GUM clinic had higher CD4 counts than men

(an average of 111 cells higher). In the Johns Hopkins study however, women had

lower CD4 counts than men at equivalent viral load levels.

Another suggestion is that active use of cocaine amongst

male members of the cohort may have boosted viral replication without affecting

the rate of disease progression. Such differences have not been detected in

other studies which have set out to look for any effect of active drug use on

disease progression.